Timothy D. Folsom

Chronic olanzapine treatment causes differential expression of genes in frontal cortex of rats as revealed by DNA microarray technique.

Viral regulation of aquaporin 4, connexin 43, microcephalin and nucleolin.

Expression of phosphodiesterase 4 is altered in the brains of subjects with autism.

Maternal infection leads to abnormal gene regulation and brain atrophy in mouse offspring: implications for genesis of neurodevelopmental disorders.

PDE4B polymorphisms and decreased PDE4B expression are associated with schizophrenia.

The role of cerebellar genes in pathology of autism and schizophrenia.

Expression of astrocytic markers aquaporin 4 and connexin 43 is altered in brains of subjects with autism.

Chronic psychotropic drug treatment causes differential expression of connexin 43 and GFAP in frontal cortex of rats.

Dopamine and serotonin levels following prenatal viral infection in mouse--implications for psychiatric disorders such as schizophrenia and autism.

GABA(A) receptor downregulation in brains of subjects with autism.

Expression of GABA(B) receptors is altered in brains of subjects with autism.

Phosphodiesterase-4A expression is reduced in cerebella of patients with bipolar disorder.

The neurodevelopmental hypothesis of schizophrenia, revisited.

Chronic psychotropic drug treatment causes differential expression of Reelin signaling system in frontal cortex of rats.

Abnormal expression of myelination genes and alterations in white matter fractional anisotropy following prenatal viral influenza infection at E16 in mice.

Prenatal viral infection of mice at E16 causes changes in gene expression in hippocampi of the offspring.

The role of lithium in modulation of brain genes: relevance for aetiology and treatment of bipolar disorder.

mRNA and protein levels for GABAAalpha4, alpha5, beta1 and GABABR1 receptors are altered in brains from subjects with autism.

Levels of phosphodiesterase 4A and 4B are altered by chronic treatment with psychotropic medications in rat frontal cortex.

Fragile X mental retardation protein levels are decreased in major psychiatric disorders.

The role of fragile X mental retardation protein in major mental disorders.

The viral theory of schizophrenia revisited: abnormal placental gene expression and structural changes with lack of evidence for H1N1 viral presence in placentae of infected mice or brains of exposed offspring.

Deficits in GABA(B) receptor system in schizophrenia and mood disorders: a postmortem study.

Dysregulation of fragile × mental retardation protein and metabotropic glutamate receptor 5 in superior frontal cortex of individuals with autism: a postmortem brain study.

Metabotropic glutamate receptor 5 upregulation in children with autism is associated with underexpression of both Fragile X mental retardation protein and GABAA receptor beta 3 in adults with autism.

Comparative gene expression study of the chronic exposure to clozapine and haloperidol in rat frontal cortex.

A Review of Varenicline's Efficacy and Tolerability in Smoking Cessation Studies in Subjects with Schizophrenia.

The involvement of Reelin in neurodevelopmental disorders.

Impairment of fragile X mental retardation protein-metabotropic glutamate receptor 5 signaling and its downstream cognates ras-related C3 botulinum toxin substrate 1, amyloid beta A4 precursor protein, striatal-enriched protein tyrosine phosphatase, and homer 1, in autism: a postmortem study in cerebellar vermis and superior frontal cortex.

Phosphorylated fragile X mental retardation protein at serine 499, is reduced in cerebellar vermis and superior frontal cortex of subjects with autism: implications for fragile X mental retardation protein-metabotropic glutamate receptor 5 signaling.

A review of traditional and novel treatments for seizures in autism spectrum disorder: findings from a systematic review and expert panel.

Downregulation of GABAA receptor protein subunits α6, β2, δ, ε, γ2, θ, and ρ2 in superior frontal cortex of subjects with autism.

Existence of monomer and dimer forms of mGluR5, under reducing conditions in studies of postmortem brain in various psychiatric disorders.

GABA receptor subunit distribution and FMRP-mGluR5 signaling abnormalities in the cerebellum of subjects with schizophrenia, mood disorders, and autism.

Protein expression of targets of the FMRP regulon is altered in brains of subjects with schizophrenia and mood disorders.

Neuropathological Mechanisms of Seizures in Autism Spectrum Disorder.

The effects of prenatal H1N1 infection at E16 on FMRP, glutamate, GABA, and reelin signaling systems in developing murine cerebellum.

GABA and GABA receptor dysregulation in superior frontal cortex of subjects with schizophrenia and bipolar disorder.

Altered subcellular localization of fragile X mental retardation signaling partners and targets in superior frontal cortex of individuals with schizophrenia.

Metabotropic glutamate receptor 5 tracer [F]-FPEB displays increased binding potential in postcentral gyrus and cerebellum of male individuals with autism: a pilot PET study.

Quantitative proteomics of forebrain subcellular fractions in fragile X mental retardation 1 knockout mice following acute treatment with 2-Methyl-6-(phenylethynyl)pyridine: Relevance to developmental study of schizophrenia.

Early Chronic Fluoxetine Treatment of Ts65Dn Mice Rescues Synaptic Vesicular Deficits and Prevents Aberrant Proteomic Alterations.

Quantitative proteomics of dorsolateral prefrontal cortex reveals an early pattern of synaptic dysmaturation in children with idiopathic autism.

Targeting the intracellular immune checkpoint CISH with CRISPR-Cas9-edited T cells in patients with metastatic colorectal cancer: a first-in-human, single-centre, phase 1 trial.