Julie Mazzolini

Localization of HIV-1 Vpr to the nuclear envelope: impact on Vpr functions and virus replication in macrophages.

Inhibition of phagocytosis in HIV-1-infected macrophages relies on Nef-dependent alteration of focal delivery of recycling compartments.

HIV-1 Nef triggers macrophage fusion in a p61Hck- and protease-dependent manner.

Single-domain antibody-SH3 fusions for efficient neutralization of HIV-1 Nef functions.

Combined loss of cRel/p50 subunits of NF-κB leads to impaired innate host response in sepsis.

Cellular entry of nanoparticles via serum sensitive clathrin-mediated endocytosis, and plasma membrane permeabilization.

The NF-κB signaling protein Bcl10 regulates actin dynamics by controlling AP1 and OCRL-bearing vesicles.

The HIV-1 protein Vpr impairs phagosome maturation by controlling microtubule-dependent trafficking.

Dynamin-Actin Cross Talk Contributes to Phagosome Formation and Closure.

Tumor initiating cells induce Cxcr4-mediated infiltration of pro-tumoral macrophages into the brain.